論文

査読有り
1993年5月

SERUM-SOLUBLE INTERLEUKIN-6 RECEPTOR IN MRL/LPR MISE IS ELEVATED WITH AGE AND MEDIATES THE INTERLEUKIN-6 SIGNAL

EUROPEAN JOURNAL OF IMMUNOLOGY
  • H SUZUKI
  • ,
  • K YASUKAWA
  • ,
  • T SAITO
  • ,
  • M NARAZAKI
  • ,
  • A HASEGAWA
  • ,
  • T TAGA
  • ,
  • T KISHIMOTO

23
5
開始ページ
1078
終了ページ
1082
記述言語
英語
掲載種別
研究論文(学術雑誌)
DOI
10.1002/eji.1830230515
出版者・発行元
VCH PUBLISHERS INC

The characteristics of soluble interleukin-6 receptor (sIL-6R) in murine sera were examined. To investigate a relationship between serum sIL-6R level and autoimmune diseases, quantitative analysis of serum sIL-6R in MRL/lpr mice was performed by an enzyme-linked immunosorbent assay. The serum sIL-6R level in MRL/lpr mice of both sexes was below the detection limit (< 1.0 ng/ml) at 8 weeks of age, but it increased in accordance with age and reached 42 +/- 9.3 ng/ml in female and 31 +/- 13 ng/ml in male mice at 30 weeks of age. In MRL/+ mice, although an age-associated increase in serum sIL-6R level was observed, it was much less extensive than that in MRL/lpr mice. Elevated serum sIL-6R level at the age of 30 weeks was observed in female and male (NZB x NZW)F1 mice (32 +/- 10 ng/ml and 17 +/- 5.0 ng/ml, respectively), and male BXSB/Mpj Yaa mice (42 +/- 18 ng/ml), suggesting that elevated serum sIL-6R in aged mice is one of the characteristics of autoimmune-prone mice. Quantitative analysis of serum IL-6 in MRL/lpr revealed that the serum sIL-6R level correlated well with the serum IL-6 level. We also showed that sIL-6R in the sera from MRL/lpr mice could mediate the IL-6 functions through the IL-6 signal-transducing receptor component gp130, suggesting that elevated production of sIL-6R may partly contribute to development of autoimmune disease in MRL/lpr mice.

リンク情報
DOI
https://doi.org/10.1002/eji.1830230515
Web of Science
https://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcAuth=JSTA_CEL&SrcApp=J_Gate_JST&DestLinkType=FullRecord&KeyUT=WOS:A1993LC38100014&DestApp=WOS_CPL
ID情報
  • DOI : 10.1002/eji.1830230515
  • ISSN : 0014-2980
  • Web of Science ID : WOS:A1993LC38100014

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