Nanako Hamada

Profile Information

Affiliation: Institute for Developmental Research,Aichi Human Service Center

Degree: 薬科学(岐阜大学)

Researcher number: 70721835
J-GLOBAL ID: 201801021435993635
researchmap Member ID: B000343689

Research Interests

Research Areas

Research History

Apr, 2019 - Nov, 2019

リサーチレジデント, 分子病態研究部, 愛知県医療療育総合センター発達障害研究所
Apr, 2016 - Mar, 2019

特別研究員PD, 日本学術振興会
Apr, 2013 - Mar, 2016

特別研究員RPD, 日本学術振興会

Education

Apr, 2010 - Mar, 2013

Gifu University

Awards

Major Papers

De novo PHACTR1 mutations in West syndrome and their pathophysiological effects.

Hamada N, Ogaya S, Nakashima M, Nishijo T, Sugawara Y, Iwamoto I, Ito H, Maki Y, Shirai K, Baba S …

Brain : a journal of neurology, 141(11) 3098-3114, Nov, 2018 Peer-reviewed
MUNC18-1 gene abnormalities are involved in neurodevelopmental disorders through defective cortical architecture during brain development

Nanako Hamada, Ikuko Iwamoto, Hidenori Tabata, Koh-ichi Nagata

ACTA NEUROPATHOLOGICA COMMUNICATIONS, 5(1) 92, Nov, 2017 Peer-reviewed
SIL1, a causative cochaperone gene of Marinesco-Sjogren syndrome, plays an essential role in establishing the architecture of the developing cerebral cortex

Yutaka Inaguma, Nanako Hamada, Hidenori Tabata, Ikuko Iwamoto, Makoto Mizuno, Yoshiaki V. Nishimura, Hidenori Ito, Rika Morishita, Motomasa Suzuki, Kinji Ohno …

EMBO MOLECULAR MEDICINE, 6(3) 414-429, Mar, 2014 Peer-reviewed

Misc.

Pathophysiological mechanism of Munc18-1 mutations in early infantile epilepsies

K-I. Nagata, N. Hamada

JOURNAL OF NEUROCHEMISTRY, 142 115-115, Aug, 2017
Rbfox1, an autism causal gene, plays an essential role in cortical development

N. Hamada, H. Ito, H. Tabata, K-I. Nagata

JOURNAL OF NEUROCHEMISTRY, 134 253-254, Aug, 2015
Abnormal cortical neuron migration by perturbed nucleus-centrosome coupling underlies the pathophysiology of autism with abnormality in RBFOX1/A2BP1 gene.

K. Nagata, N. Hamada, H. Ito, I. Iwamoto, R. Morishita, H. Tabata

MOLECULAR BIOLOGY OF THE CELL, 26, 2015
発達障害の背景としての大脳皮質構築異常

浜田奈々子, 稲熊裕, 永田浩一

生化学, 87(2) 205-208, 2015 Peer-reviewedInvited
Biochemical and morphological characterization of an autism-related molecule, A2BP1, in developing cerebral cortex

K-I. Nagata, N. Hamada, H. Ito, I. Iwamoto, M. Mizuno, R. Morishita, Y. Inaguma, H. Tabata

MOLECULAR BIOLOGY OF THE CELL, 24, 2013
SIL1, a causative gene of Marinesco-Sjogren syndrome, plays an essential role in establishing the architecture of the developing cerebral cortex

K-I. Nagata, Y. Inaguma, Y. Nishimura, H. Ito, M. Suzuki, N. Hamada, M. Mizuno, I. Iwamoto, R. Morishita, T. Kumagai

MOLECULAR BIOLOGY OF THE CELL, 23, 2012

Presentations

発達障害責任遺伝子MED13Lの神経発達における役割と遺伝子変異がもたらす病態機能解析

浜田奈々子, 永田浩一

第53回日本臨床分子形態学会学術集会, Oct 22, 2021
De novo PHACTR1 mutations in West Syndrome and their pathophysiological effects.

浜田奈々子

The 20th Annual Meeting of Infantile Seizure Society, May 31, 2019
ウエスト症候群の新規責任遺伝子の同定と病態機能解析（推薦口演）

浜田奈々子

第41回小児遺伝学会学術集会, Jan 11, 2019
小児難治性てんかん責任遺伝子の病態生理解析

浜田奈々子

2018年度生理学研究所研究会「神経発達・再生研究会」, Oct 18, 2018
Identification of PHACTR1 as a novel causal gene for West Syndrome and pathophysiological significance of the gene mutations

浜田奈々子

第61回日本神経化学会大会, Sep 9, 2018
Role of Munc18-1 in cortical neuron migration

浜田奈々子

第90回日本生化学会大会, Dec 7, 2017
Munc18-1 plays an essential role for cortical neuron migration during brain development

浜田奈々子

第60回日本神経化学会大会, Sep 8, 2017
Pathophysiological role of MUNC18-1 in early infantile epilepsies

浜田奈々子

第9回名古屋グローバルリトリート, Feb 10, 2017 Invited
Essential role of Munc18-1 in neuronal migration during cortical development

浜田奈々子

第8回名古屋グローバルリトリート, Feb 12, 2016
Rbfox1, an autism causal gene, plays an essential role in cortical development

浜田奈々子

25th International Society for Neurochemistry, Aug 26, 2015

Professional Memberships

Research Projects

Phactr1遺伝子変異を背景とするウエスト症候群の分子病態メカニズムの解明

科学研究費助成事業, 基盤研究(C), 日本学術振興会, Apr, 2020 - Mar, 2023

浜田奈々子

Profile Information

Research Interests

Research Areas

Research History

Education

Awards

Major Papers

Misc.

Presentations

Professional Memberships

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