Michael R. Bowl, Michelle M. Simon, Neil J. Ingham, Simon Greenaway, Luis Santos, Heather Cater, Sarah Taylor, Jeremy Mason, Natalja Kurbatova, Selina Pearson, Lynette R. Bower, Dave A. Clary, Hamid Meziane, Patrick Reilly, Osamu Minowa, Lois Kelsey, The International Mouse Phenotyping Consortium, Glauco P. Tocchini-Valentini, Xiang Gao, Allan Bradley, William C. Skarnes, Mark Moore, Arthur L. Beaudet, Monica J. Justice, John Seavitt, Mary E. Dickinson, Wolfgang Wurst, Martin Hrabe de Angelis, Yann Herault, Shigeharu Wakana, Lauryl M. J. Nutter, Ann M. Flenniken, Colin McKerlie, Stephen A. Murray, Karen L. Svenson, Robert E. Braun, David B. West, K. C. Kent Lloyd, David J. Adams, Jacqui White, Natasha Karp, Paul Flicek, Damian Smedley, Terrence F. Meehan, Helen E. Parkinson, Lydia M. Teboul, Sara Wells, Karen P. Steel, Ann-Marie Mallon & Steve D. M. Brown - Show fewer authors
新規Rb1変異発がんモデルマウスにおける下垂体腫瘍発生と甲状腺腫瘍抑制の機構(A novel retinoblastoma mutation predisposed mice to pituitary tumors but led to selective suppression of thyroid tumors)
Serca2に新規点変異を持つマウスに発症する消化管腫瘍は、その変異部位により異なる速さで進展する(Differential development of epithelial cell tumors in mouse alimentary tract caused by Serca2 novel allelic mutations)
