MISC

2009年5月

Autophagy regulates progression of programmed cell death during petal senescence in Japanese morning glory

AUTOPHAGY
  • Kenichi Shibuya
  • ,
  • Tetsuya Yamada
  • ,
  • Kazuo Ichimura

5
4
開始ページ
546
終了ページ
547
記述言語
英語
掲載種別
DOI
10.4161/auto.5.4.8310
出版者・発行元
LANDES BIOSCIENCE

Petal senescence is a type of programmed cell death (PCD) that is tightly regulated by multiple genes. We recently reported that a putative membrane protein, InPSR26, regulates progression of PCD during petal senescence in Japanese morning glory (Ipomoea nil). Reduced InPSR26 expression in transgenic plants (PSR26r lines) resulted in accelerated petal senescence with hastened development of PIED symptoms, and transcript levels of autophagy-related genes were reduced in the petals. Autophagy visualized by monodansylcadaverine staining indicated reduced autophagic activity in the PSR26r plants. The results from our recent studies suggest that InPSR26 acts to delay the progression of PCD during petal senescence possibly through regulation of the autophagic process. In this addendum, we discuss the role of autophagy in petal senescence as it relates to these findings.


リンク情報
DOI
https://doi.org/10.4161/auto.5.4.8310
PubMed
https://www.ncbi.nlm.nih.gov/pubmed/19337027
Web of Science
https://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcAuth=JSTA_CEL&SrcApp=J_Gate_JST&DestLinkType=FullRecord&KeyUT=WOS:000266118900017&DestApp=WOS_CPL
URL
https://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=66349119967&origin=inward
ID情報
  • DOI : 10.4161/auto.5.4.8310
  • ISSN : 1554-8627
  • PubMed ID : 19337027
  • SCOPUS ID : 66349119967
  • Web of Science ID : WOS:000266118900017

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