SABE Hisataka

J-GLOBAL         Last updated: Sep 12, 2018 at 11:54
 
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Name
SABE Hisataka
URL
http://g21001.med.hokudai.ac.jp/index-e.html
Affiliation
Hokkaido University
Section
Graduate School of Medicine
Job title
Professor
Degree
(BLANK), (BLANK)

Research Areas

 
 

Academic & Professional Experience

 
1986
   
 
Instructor, Kyoto University School of Medicine
 
1990
   
 
The Rockefeller University Laboratory of
 
 
   
 
Molecular Oncology Postdoctoral Associate
 
1993
   
 
The Rockefeller University Laboratory of
 
 
   
 
Molecular Oncology Assistant Professor
 

Education

 
 
 - 
1986
Graduate School, Division of Medicine, Kyoto University
 
 
 - 
1980
Faculty of Science, Kyoto University
 

Published Papers

 
Otsuka Y, Oikawa T, Yoshino H, Hashimoto S, Handa H, Yamamoto H, Hashimoto A, Sabe H
Cell communication and signaling : CCS   16(1) 1   Jan 2018   [Refereed]
Oikawa T, Otsuka Y, Onodera Y, Horikawa M, Handa H, Hashimoto S, Suzuki Y, Sabe H
Scientific reports   8(1) 1595   Jan 2018   [Refereed]
Tien DN, Kishihata M, Yoshikawa A, Hashimoto A, Sabe H, Nishi E, Kamei K, Arai H, Kita T, Kimura T, Yokode M, Ashida N
Scientific reports   4 5094   May 2014   [Refereed]
Mazaki Y, Onodera Y, Higashi T, Horinouchi T, Oikawa T, Sabe H
Cell communication and signaling : CCS   15(1) 36   Oct 2017   [Refereed]
Erythrocyte protein bond 4.1-like5 expression in patients with upper urinary tract urothelial carcinoma correlates with tumor recurrence
Daimon Tatsuaki, Kosaka Takeo, Mikami Shuji, Miyazaki Yasumasa, Mizuno Ryuichi, Kikuchi Eiji, Miyajima Akira, Nakagawa Ken, Sabe Hisataka, Oya Mototsugu
JOURNAL OF CLINICAL ONCOLOGY   32(15)    May 2014   [Refereed]

Misc

 
真崎雄一, 小野寺康仁, 東恒仁, 堀之内孝広, 及川司, 佐邊壽孝
日本細胞生物学会大会(Web)   69th ROMBUNNO.T8‐11(P1‐077) (WEB ONLY)   2017
古川聖太郎, 橋本あり, 橋本茂, 小野寺康仁, 及川司, 大塚勇太郎, 佐邊壽孝, 平野聡
日本外科学会定期学術集会(Web)   116th PS-002-2 (WEB ONLY)-002   Apr 2016
Y. Komiya, Y. Onodera, M. Kuroiwa, S. Nomimura, Y. Kubo, J. M. Nam, J. M. Nam, K. Kajiwara, S. Nada, C. Oneyama, H. Sabe, M. Okada
Oncogenesis   5    Sep 2016
© 2016 The Author(s). Epithelial tumor cells often acquire malignant properties, such as invasion/metastasis and uncontrolled cell growth, by undergoing epithelial-mesenchymal transition (EMT). However, the mechanisms by which EMT contributes to m...
小野寺康仁, 堀川芽衣, 佐邊壽孝
がんと代謝研究会プログラム&抄録集   4th 44   Jul 2016
p53はEZH2と機能的に競合することで上皮性維持に寄与する
及川 司, 大塚 勇太郎, 小野寺 康仁, 半田 悠, 橋本 あり, 橋本 茂, 鈴木 穣, 佐邊 壽孝
日本癌学会総会記事   75回 J-3030   Oct 2016

Books etc

 
Properties of human IL2 receptor expressed on non-lymphoid cells by cDNA transfection.(共著)
Mol.Biol.Med.   1984   
Interleukin 2 receptor ; structure, function, and expression.(共著)
Cold Spring Harbor Symposia on Quantitative Biology   1986   
Activation of c-Src in cells bearing v-Crk and its suppression by Csk.(共著)
Mol.Cell.Biol.   1992   
Analysis of Csk SH2 binding to tyrosine phosphorylated proteins in c-Src suppression and mitotic activation.(共著)
Proc.Natl.Acad.Sci.USA   1994   
Growth factors and receptors of lymphocytes.(共著)
Acta Neurochiurgica (eds.K.Sano and S.Ishii, Springer-Verlag, NY, USA)   1987   

Conference Activities & Talks

 
The Arf6 pathway: a central pathway driving mesenchymal malignancies and drug-resistance of refractory cancers [Invited]
SABE Hisataka
7 Jul 2017   
Hisataka Sabe, Shigeru Hashimoto, Ari Hashimoto, Yutaro Otsuka, Haruka Handa, Tsukasa Oikawa, and Yasuhito Onodera
15 Jun 2016   
Hisataka Sabe, Ari Hashimoto, Shigeru Hashimoto, Hirokazu Sugino, Ayumu Yoshikawa, Haruka Handa, Yutaro Otsuka, Tsukasa Oikawa, Shuji Mikami, Mototsugu Oya
4 Nov 2014   
SABE Hisataka
3 Oct 2013   

Association Memberships

 
 

Research Grants & Projects

 
Ministry of Education, Culture, Sports, Science and Technology: Grants-in-Aid for Scientific Research(基盤研究(A))
Project Year: 2008 - 2011    Investigator(s): Hisataka SABE
(抄録なし)
Ministry of Education, Culture, Sports, Science and Technology: Grants-in-Aid for Scientific Research(特定領域研究)
Project Year: 2005 - 2009    Investigator(s): Hisataka SABE
Most malignant human tumors are epithelial cell origin. The basic fear of such tumors are based on their invasion and metastasis. Activation of integrins and inactivation of E-cadherin are hallmark characteristics for such invasive and metastatic ...
Ministry of Education, Culture, Sports, Science and Technology: Grants-in-Aid for Scientific Research(基盤研究(B))
Project Year: 2006 - 2007    Investigator(s): Hisataka SABE
We aim to elucidate mechanisms controlling cell migration and the polarity formation, via investigating their relationship to intracellular vesicle trafficking. Our other main research interest is to understand the principal mechanisms involved in...
Ministry of Education, Culture, Sports, Science and Technology: Grants-in-Aid for Scientific Research(基盤研究(B))
Project Year: 2004 - 2005    Investigator(s): Hisataka SABE
Cell migration is a multifactorial process in which a number of distinct events occur simultaneously. The major purpose of this study is to understand basic molecules and mechanisms coordinately regulating cell migration and invasion.Arf6 plays es...
Ministry of Education, Culture, Sports, Science and Technology: Grants-in-Aid for Scientific Research(基盤研究(B))
Project Year: 2002 - 2003    Investigator(s): Hisataka SABE
Previously we reported that AMAP2/PAG3/Papα/KIAAO400, a GTPase-activating protein (GAP), acts to antagonize Arf6 function when overexpressed, while it was shown to exhibit efficient GAPing activities for other Arf isoforms in vitro. During this pe...