論文

査読有り
2014年1月

To be or not to be: regulation of restriction-modification systems and other toxin-antitoxin systems

NUCLEIC ACIDS RESEARCH
  • Iwona Mruk
  • ,
  • Ichizo Kobayashi

42
1
開始ページ
70
終了ページ
86
記述言語
英語
掲載種別
研究論文(学術雑誌)
DOI
10.1093/nar/gkt711
出版者・発行元
OXFORD UNIV PRESS

One of the simplest classes of genes involved in programmed death is that containing the toxin-antitoxin (TA) systems of prokaryotes. These systems are composed of an intracellular toxin and an antitoxin that neutralizes its effect. These systems, now classified into five types, were initially discovered because some of them allow the stable maintenance of mobile genetic elements in a microbial population through postsegregational killing or the death of cells that have lost these systems. Here, we demonstrate parallels between some TA systems and restriction-modification systems (RM systems). RM systems are composed of a restriction enzyme (toxin) and a modification enzyme (antitoxin) and limit the genetic flux between lineages with different epigenetic identities, as defined by sequence-specific DNA methylation. The similarities between these systems include their postsegregational killing and their effects on global gene expression. Both require the finely regulated expression of a toxin and antitoxin. The antitoxin (modification enzyme) or linked protein may act as a transcriptional regulator. A regulatory antisense RNA recently identified in an RM system can be compared with those RNAs in TA systems. This review is intended to generalize the concept of TA systems in studies of stress responses, programmed death, genetic conflict and epigenetics.

リンク情報
DOI
https://doi.org/10.1093/nar/gkt711
PubMed
https://www.ncbi.nlm.nih.gov/pubmed/23945938
Web of Science
https://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcAuth=JSTA_CEL&SrcApp=J_Gate_JST&DestLinkType=FullRecord&KeyUT=WOS:000331136000013&DestApp=WOS_CPL
ID情報
  • DOI : 10.1093/nar/gkt711
  • ISSN : 0305-1048
  • eISSN : 1362-4962
  • PubMed ID : 23945938
  • Web of Science ID : WOS:000331136000013

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