Keiji Wada

J-GLOBAL         Last updated: Sep 26, 2018 at 13:45
 
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Name
Keiji Wada
Affiliation
National Center of Neurology and Psychiatry
Section
National Institute of Neuroscience,Ncnp

Research Areas

 
 

Published Papers

 
Suzuki M, Sango K, Wada K, Nagai Y
Neurochemistry international   119 97-106   Oct 2018   [Refereed]
Hatanaka Y, Watase K, Wada K, Nagai Y
Scientific reports   5 16102   Nov 2015   [Refereed]
Furuta A, Kikuchi H, Fujita H, Yamada D, Fujiwara Y, Kabuta T, Nishino I, Wada K, Uchiyama Y
The American journal of pathology   185(6) 1713-1723   Jun 2015   [Refereed]
Takahashi M, Contu VR, Kabuta C, Hase K, Fujiwara Y, Wada K, Kabuta T
RNA biology   14(11) 1534-1543   Nov 2017   [Refereed]
Takeuchi F, Komaki H, Yamagata Z, Maruo K, Rodger S, Kirschner J, Kubota T, Kimura E, Takeda S, Gramsch K, Vry J, Bushby K, Lochmüller H, Wada K, Nakamura H
Neuromuscular disorders : NMD   27(10) 894-904   Oct 2017   [Refereed]

Research Grants & Projects

 
Ministry of Education, Culture, Sports, Science and Technology: Grants-in-Aid for Scientific Research(基盤研究(A))
Project Year: 2007 - 2010    Investigator(s): Keiji WADA
Ministry of Education, Culture, Sports, Science and Technology: Grants-in-Aid for Scientific Research(基盤研究(B))
Project Year: 2006 - 2008    Investigator(s): 内藤幸雄, Furusaka MICHIHIRO
Ministry of Education, Culture, Sports, Science and Technology: Grants-in-Aid for Scientific Research(基盤研究(B))
Project Year: 2004 - 2006    Investigator(s): Keiji WADA
We previously identified that ubiquitin C-terminal hydrolase Ll (UCH-L1) is the responsible gene product for the gracile axonal dystrophy (gad) phenotype (Nature Genetics, 1999). UCH-L1 is a member of the deubiquitinating enzyme family and is sele...
Ministry of Education, Culture, Sports, Science and Technology: Grants-in-Aid for Scientific Research(基盤研究(C))
Project Year: 2004 - 2005    Investigator(s): Mami NODA
Bradykinin (BK) has been reported as a mediator of brain damage in acute insults. We had found that receptors for BK were identified on microglia, the pathologic sensors of the brain (Noda et al., 2003). Here we report that BK has two functions vi...
Ministry of Education, Culture, Sports, Science and Technology: Grants-in-Aid for Scientific Research(基盤研究(B))
Project Year: 2001 - 2003    Investigator(s): Keiji WADA
We previously identified that ubiquitin C-terminal hydrolase L1 (UCH-L1) was not expressed in the gracile axonal dystrophy (gad) mouse. The gad mouse is pathologically characterized by dying-back type of axonal degeneration, and thus the mutant ap...